Reported Characteristics

Drug Class: Thyroid hormone stimulator. (Oral) Average

Reported Dosage: 20 MG (pure extract) 3-4 times daily.

Significant stimulation of thyroid function during use. Guggulsterone are an extract of the herb Commiphora Mukul, clinically proven to restore lagging metabolic rate caused by dietary calorie deficiency such as during diet phases. Ayurvedic medicine has utilized the unprocessed herb for centuries to treat energy deficiencies related ailments. Guggulsterones Z and E seem to have the greatest effect upon stimulation of thyroid hormone, though other substrates of the herb commiphora mukul may effect different metabolic factors as well. Thus far, research suggests this is a result of guggulsterones Z & E stimulation of TSH (Thyroid-Stimulating-Hormone) production. This results in an increase in Thyroid gland T-4 production and subsequent liver conversion to the more active T-3 hormone. Guggulsterones can be very beneficial to dieting athletes. When calorie intake is decreased, the body naturally decreases metabolic rate or calorie expenditure. This is why dieters often hit a fat loss wall after 2-4 weeks. Since TSH production begins the entire thyroid hormone cascade, guggulsterones can be very helpful at restoring “normal” metabolic rate at this point. It should be noted that guggulsterones do not inhibit normal thyroid activity or induce a negative feed-back loop. It is very unlikely that the use of this extract will lead to above normal accepted TSH, T-4, T-3 reference range production. Other benefits of guggulsterone supplementation include reduced blood cholesterol and triglyceride levels, increased fat oxidation (fat burning), increased energy, and better post-training recovery during diet restriction phases. Reduced acne has been noted. Please note that other diet defiencies can lead to reduced metabolic rate including phosphates, niacin, selenium, and magnesium among others, such as Zinc. Some athletes used guggulsterones to regenerate thyroid function after discontinuance of synthetic T-3/T-4 protocols. Those who thought ahead, utilized guggulsterones ” synthetic T-3/T-4 thyroid hormone use to prevent thyroid function suppression even temporarily. This usually was initiated 10-14 days prior to synthetic thyroid administration discontinuance. Coleus Forskohlii, the amino acid tyrosine, and iodine also had a noted great deal of value for this purpose.

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Drug Class: Herbal thyroid stimulator. (Oral)

Average Reported Dosage: (Standardized for 10% forskolin) 165-250 MG 3-4 times daily.

Significant stimulation of thyroid gland and adenylate cyclase production. Why is an herbal supplement in a book intended to report upon the things hardcore athletes have done? It should be recalled that most AAS synthesis begins with a plant extract as does ephedrine, methamphetamine, and heroin. The point being that powerful things come from many places…sometimes. Coleus Forskohlii increases cyclic AMP (cAMP) in cells and up-regulates thyroid gland function. Why is cellular cAMP so cool? The active substrate in Coleus Forskohlii is a diterpene derivative called forskolin. Well, forskolin stimulates the production of an enzyme called Adenylate-Cyclase (AC) which is sort of a master enzyme in the body that positively effects many other enzymes that regulate muscle growth and fat loss. In this case, AC increases cAMP which in turn activates Protein- Kinase (PK). This event allows a PK/phosphorylation reaction resulting in the active form of Hormone-Sensitive-Lipase (HSL). Finally …HSL stimulates the release of fatty acids from adipose tissue (fat cells) so muscle cells can use them as an energy (ATP) and fuel for heat source. Remember, UCP-3’s earlier in this section? Well, forskolin stimulates thyroid gland activity similarly to Thyroid-Stimulating-Hormone (TSH). TSH is also sometimes called thyrotropin. When the thyroid gland stimulated it begins the thyroid hormone cascade by releasing T-4 and so on. Since this results in an increase in circulatory thyroids hormone levels and an increase in (yup!) UCP-3’s, more of that newly released fatty acids supply is burned off.

The reason the basic biochemistry was explained here is simple. First, anything that affects any of the biochemicals we briefly discussed, also effects body composition. This includes all thermalgenics, AAS, insulin, GH, and most other anabolic chemistries. Second, the basic knowledge may prevent the reader from falling for supplement ad scams by knowing why something will or will not actually have value. Most products containing Coleus Forskohlii should be (but rarely actually are) standardized for a 10% forskolin content. An effective dosage of the 10% standardized product has been 165-250 MG, 3-4 times daily. So far there is some existing research that supports the listed effects of Coleus Forskohlii. And anecdotal /personal evaluations are positive thus far. It seems finding an actual standardized source is the most difficult aspect of acquiring favorable results. A note of interest. The use of Coleus Forskohlii seems to have possible cardiovascular benefits by acting as a vasodilator (lowers blood pressure) , inhibition of platelet aggregation (reduced blood clotting), and positive inotropic activity in the heart (increased contractile force). The use of Coleus Forskohlii does not seem to inhibit endogenous TSH production. There is a synergy between this product and beta-andrenergic drugs such as Clenbuterol, ephedrine, synephrine, and norephedrine.

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Reported Characteristics
Active-Life: About 6-8 hours (Diuretic effects)
Drug Class: Loop Diuretic (Oral)
Average Reported Dosage: 40-100mg total in a 12 hour period
Acne: None
Water Retention: You must be joking!
High Blood Pressure: None
Liver Toxic: Unknown
Aromatization: Does not apply

Lasix is probably one of the most dangerous drugs some pre-contest bodybuilders used. Even the IFBB tests for diuretics, and that is saying something Lasix was used to excrete excess water during the last day or two before a show. This was said to be especially effective with subcutaneous water (under the skin) so a “ripped to the bone, dry, and hard” look was achieved. Lasix was also used by some to lose weight for weight class oriented sports. Loop diuretics work by increasing the excretion of electrolytes (sodium, chloride, potassium) which the body normally uses to maintain intra cellular and extracellular water. Monitoring of the re-absorption of potassium, sodium, and chloride ions during use should have been a must, but few did so. Lasix can and has caused death. As to dosages, it depended upon the effect achieved. Normally 20-40-mg was taken and (over a periods of 2-4 hours) the bodybuilder evaluates the results. This was followed once or twice more at 4 hour intervals if more water loss was necessary. Too much water loss makes the body appear flat with no vascularity and makes it impossible to pump-up before walking out onto the stage. The effects of Lasix begin within about an hour of administration and continue for 6-8 hours. Once “the look” was achieved, it was unnecessary to ingest more to maintain “the look”.

Lasix is very strong and was reported to cause diarrhea, dehydration, dizziness, muscle cramps, circulatory disorders, vomiting, circulatory collapse, fainting, and cardiac arrest. It was considered far more safe to start with 20-mg and repeat every 4 hours than to use higher dosages for a shorter period of tome. Over 40-mg per dosage increased side effects dramatically!

DIUNRAL 5,10,20,40,250,500-MG TABS
DIURAPID 40/500 40,500-MG TABS

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Reported Characteristics
Active-Life: 10-12 hours
Drug Class: Diuretic (Oral)
Average Reported Dosage: 50-200mg (total) daily

Spironothiazid is a diureteic and an ALDOSTERONE antagonist. This means that it suppresses the water retaining actions of the hormone ALDOSTERONE while lowering water retention by lowering electrolytes (potassium, sodium, and calcium). The advantage of the combination is that the potassium absorption effect by the spironolactone can be mediated by the hydrochlorthiazide. Because of this, some of the potassium loss side effects can be avoided. This is probably a safer choice than LASIX though any diuretic use should be medically monitored. Thiazides also lead to a lower loss of calcium. Side effects are commonly due to imbalances in electrolytes and fluids. They can include irregular pulse rate, cramps, and light headiness. Since the drug has ANTIANDROGENIC quality (spironolactone), men can experience possible gyno, and impotence due to higher dosage and/or prolonged use. These were noted to not occur due to the brief administration period normally employed. According to available literature, it is best to avoid higher potassium intake during use. Dosages were usually 50-150mg a day for no more than 3 days. Dosages were divided into 2-3 daily dosages.

Aldactazide 25/15-MG TABS
Aldactazide 25/50-MG TABS
Spironothiazid 25/25-MG TABS



*The most important water store is in blood and the vascular system. Without adequate water in the vascular system blood volume is compromised, and if severe enough, the result is death. So this rates a big number one in the water store hierarchy. *The second on the big three list is muscle tissue. Water is required within all muscle tissues, both smooth and fibrous, to support life sustaining metabolic processes. *The last area of importance for water storage is subcutaneous (under the skin) areas. For those who have not been paying attention, this is the area that a bodybuilder wants to eliminate as much water from as possible the day of a show. The results are a “make-it or break-it” issue. So how do we do it? Piece of cake! (But not during contest dieting!) The key to subcutaneous water control depends upon control of the hormone Aldosterone. Obviously estrogen control is part of this hormone cascade action/reaction factor.

But, our main focus is salt and water control, so Aldosterone is the key. Beginning 15 days out from a show, an athlete should increase salt intake 20- 30%. This of course means salt intake was never reduced to begin with. The amount must remain reasonably high and steady each day. This creates an environment in which the body does not have to release Aldosterone. This causes salt to stay in muscle tissue and the subsequent attraction of water stores there. Also, the all important maintaining of the sodium-potassium pump is accommodated as well. (During diet phases, this also reduces catabolism.) During the 15 day period, water intake must absolutely remain high. 1.5-2.0 gallons daily is a base line in fact. This helps your body excrete any extra sodium, which of course it will, because Aldosterone secretion in the body has been controlled by elevated salt intake/water intake. The body will continue to dump all excess water and sodium as long as this is followed. On the Friday night before a Sunday show, the athlete stops water intake. The body thinks it will still get the 1.5-2.0 gallons of water daily and continues to excrete water at its normal rate. This causes a decrease in blood volume and of course muscle water volume. Remember the body’s water hierarchy? Well, as a survival response or reaction, the body gives up water from the area of least importance as a means of compensation. Yup, you got it. Subcutaneous water is pumped into blood and muscles. The result is vascularity, full muscle bellies, and paper thin skin. *It’s always a matter of working with, not against the body’s action/reaction factors to accomplish the greatest progress and /or results. *This works well with Creatine/Dextrose carb-loading also.

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Reported Characteristics
Active-Life: 8-12 hours
Drug Class: Steroid Biosynthesis Inhibitor (oral)
Average Reported Dosage: 500mg-2000mg Daily
Acne: None
Water Retention: None
High Blood Pressure: Unknown
Noted Comments: Powerful endogenous hormone inhibitor
Aromatization: None
Liver Toxic: High potential


Aminoglutethimide is normally prescribed for patients with breast cancer or Cushings Syndrome. In both cases patients suffer from catastrophic physiological decline. To a profound degree this is notably due to severe over production of endogenous glucocorticoids. The one glucocorticoid most readers would be familiar with is cortisol. Bodybuilders and strength athletes have often utilized this drug as a means of inhibiting the P-450 and aromatase enzymes. This obviously refers to inhibition of the two potential pathways in which susceptible AAS are converted into estrogens. The result of administration of aminoglutethimide is partial or complete inhibition of endogenous hormone biosynthesis. When this drug is introduced into the body it blocks the conversion of cholesterol into pregnenolone. Since this is the first step in all hormone biosynthesis: By blocking the conversion of C-19 androgens into C-15 estrogens it functions as an anti-estrogen. This is one of the obvious intents reported users had in mind. But by inhibiting the conversion of cholesterol into pregnenolone it also proportionately inhibits the endogenous biosynthesis of all hormones including androgens, estrogens, aldosterone, and cortisol. This brings the discussion to the second reason this drug use was commonly reported. Cortisol is a catabolic hormone that just loves to eat muscle tissue.


In fact it is a significant part of the equation that induces genetic limitations to muscular augmentation. Many authorities and athletes alike have realized on a few facts: (1) Less cortisol = less muscle catabolism (destruction) (2) Less estrogen = less fat accumulation and less gyno resulting in a leaner physique (3) Less aldosterone = less water retention and a harder appearance. But less conversion of cholesterol into pregnenolone = less endogenous testosterone. Most noted these effects were highly desirable since the absence of natural testosterone production was abundantly replace through AAS use. And it was generally acknowledged that the absence of competing hormones had a profound synergistic effect upon those the selfadministered. According to the available returns the average reported dosages were 500-750mg daily to inhibit excessive estrogen formation from AAS aromatization and 1000-2000mg daily for the purpose of cortisol inhibition. Most reported a scaled weekly dosage increase was most effective: Week #1 250mg 2xd, week #2 250mg 3xd, week #3 & 4 500mg 2xd. *My personal experience with this drug was that a 2 day on – 2 day off protocol worked best with fewer negative side effects. Of the many side effects is that the drug inhibits the body’s ability to react to inflammatory responses. This means it can prevent the body from inhibiting hemorrhaging (I do hope that no one cut themselves shaving!) and fight disease among other things. It can also make a bodybuilder a victim of CUSSING SYNDROME. Other side effects include: Sore joints from a reduction in glucocorticoids, reduced white/red blood cell counts, reduced platelet counts, and liver disease. The package insert states 2-7 250-mg tabs daily for treatment of CUSHINGS SYNDROME. CUSHINGS victims have a “much” higher cortisol/cortisone production than even the most over trained athlete. For this reason 500 mg-1000 mg daily total was considered more than enough for a chemically enhanced bodybuilder for cortisol/cortisone suppression, and 250 mg -500 mg sufficient for use as an anti-estrogen and aldosterone control drug during cycles. It was also noted that Aminoglutethmide was not be utilized for more than 4-6 weeks. At that point, the body responded by increasing production of ACTH and a whole new series of catabolic effects resulted.







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Reported Characteristics
Active-Life: 12-24 hours oral
Drug Class: Anti-estrogen/Aromatase enzyme inhibitor
Average Reported Dosage: Oral: 200-300mg daily; Injectable 100mg/1ml daily
Acne: Rare
Water Retention: None
High Blood Pressure: Rare
Liver Toxic: Not listed but unlikely
DHT Conversion: None
Decreases HPTA Function: No. (Actually increased HPTA activity)
Aromatization: None

Teslac is used as an anti-estrogen. Instead of acting as an estrogen antagonist (competing for estrogen receptor-sites), Teslac prevents the aromatization of androgens into estrogens by inhibiting the aromatase enzyme. Obviously this prevents or minimizes the negative effects of estrogen such as gyno, water retention, and female pattern fat deposits. Teslac is unique in that it is reputed to cause permanent irreversible suppression of estrogen production in males. Technically speaking, Teslac is an oral androgenic steroid related to testosterone. However, it has only very low androgenic and no anabolic effect. Medical use of Teslac is for treatment of breast carcinoma in women. For male athletes, it is probably the best anti-estrogen available, and it causes an increase in natural (endogenous) testosterone production. Teslac elevates natural testosterone production by influencing the HYPOTHALAMUS to stimulate the PITUITARY to release more gonadotropin. This in turn simulates the testes Leydig cells to produce more testosterone at a significantly increased level (Comparable to HCG). Unlike HCG, Teslac takes several days to cause this effect.

So administration over a prolonged period (several days) clinically considered necessary (under a doctors care). For this reason Teslac does “create” an improved anabolic/androgenic environment. Even so-called natural (like lifting tons of weight and drinking protein powders is natural) bodybuilders have employed benefits form this drug. *Plasma calcium elevation has been during administration of Teslac. Manufacturer inserts suggest a dosage of 250-1000mg daily. However, this is not reported as necessary for estrogen suppression. Normally 200-300mg daily divided into 2-3 doses was reported as quite effective. Some reported 100-mg of the injection form version was even more effective. Due to the high price of Teslac, many have stacked it with PROVIRON: Teslac-100-mg daily and Proviron 50-mg daily. Women users reported excellent hardening effects with few, if any, side effects such as virilization.

Side effects are very rare but could include nausea, vomiting, tongue infection, swelling and pain in arms and legs, prickling sensation and high blood pressure, as well as loss of appetite. As mentioned earlier, side effects were rare but women often skipped menstruation during use. This is probably rated as the most effective overall anti-estrogen available whether during or post cycle as well as for pre-contest overall hardening effects.


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Reported Characteristics
Active-Life: 64 hours
Drug Class: leutenizing Hormone (LH) – Gonadotropin
Average Reported Dosage: Men 2000-5000 i.u. injected every 5th day
Acne: Yes
Water Retention: Yes, HCG is a female hormone.
Liver Toxic: None
Aromatization: None, however due to HCG use testosterone levels increased and
aromatize was a potential side effect

HCG is used medically to positively influence ovulation in women, and to help produce estrogen. It is also utilized in fertility medicine to aid in ovulation. Male bodybuilders used HCG for another important reason. HCG is almost exactly the same amino acid sequence as Luteinizing Hormone (LH). LH is normally produced in the pituitary gland which is then circulated to the testes where it contacts the Leydig cells. The Leydig cells then produce androgens such as testosterone. Obviously this means so-called natural bodybuilders reported use of HCG to increase endogenous (natural) testosterone levels. According to some clinical studies this works so well that an injection of 1500-2000 i.u. of HCG has increased plasma testosterone levels 200-300% over normal levels. For those males who utilized high aromatizing AAS, HCG was a “partial” cure for restarting natural testosterone production either mid or post-cycles. When administering exogenous (occurring outside the body) androgens, such as AAS, the body’s endocrine system shuts down (partially of completely) natural androgen production in an attempt to maintain homeostasis.

This is simply because the hypothalamus senses the excess estrogens from AAS aromatization (and to a lesser degree elevated androgen levels) and signals the pituitary (hypophysis) to partially or completely stop producing LH and FSH (follicle stimulating hormone). Since the Leydig cells in the testes do not receive the signal from LH, they partially or completely shut down testosterone production. Sperm production is also reduced as a result of FSH downregulation. Because the testes are not producing androgens and/or sperm at their normal level, the testes shrink. This often causes a drop in libido too. Many heavy AAS users reported sexual dysfunction for a brief (Brief?) period post-cycle. So what did the men do about “the boy’s”? HCG injections act as a replacement for the LH normally produced by the pituitary gland which normally stimulates androgen and sperm production by the testes. When 2000-5000 i.u. of HCG was injected every 5th day for 2-3 weeks, mid or post-cycle, the testes began to function again.

Also an increase in total testosterone was realized and athletes often made some of their best gains at this point. It also helped to keep the “significant other” significantly happy! When HCG was administered beginning the last week of an AAS cycle and for an additional 1-2 weeks post-cycle, testes function normalized again and much of the common post- AAS cycle muscle mass and strength loss was avoided. However, our athletes were not out of the woods (with acceptable wood) quite yet. Earlier, I mentioned that HCG has been a utilized as a “partial cure” for the shut-down of the hypothalamus-pituitary-testes-axis (HPTA). HCG only “replaces” natural LH. The pituitary and hypothalamus part of the HPTA still sense no reason to produce gonadotropins and restore normal LH/FSH production. So ending HCG administration sometimes only brought on another crash. But staying on HCG for more than 3 weeks without at least a month off between HCG cycles could cause permanent gonadal dysfunction and/or a desensitizing of Leydig Cells. Male bodybuilders commonly used Clomid or Cyclofenil with HCG (*See Clomid for more info).

Available literature shows that Clomid stimulates the pituitary to release more gonadotropin so a quicker and elevated level of LH and FSH are realized. By following an AAS cycle with 2000-5000 i.u. HCG every 5th day for 2-3 weeks and ingesting Clomid for the last 10-15 days of HCG administration many athletes noted that muscle mass and strength losses post AAS cycle were significantly avoided. Many athletes also used Clenbuterol at this point. It should be noted that administration of HCG will increase plasma testosterone levels 200-300% or more. Therefore all of the negative side effects of testosterone injections can apply to a lesser degree.

A.P.I. 5000I,U., 10,000 I,U, 20,000 I,U, AMPULE
CHORAGON 1500 I.U., 5000 I.U. AMPULE
CHOREX 5000-I.U., 10,000 I.U. AMPULE
CHORON 10 1000 I.U., 10,000 I.U. AMPULE
GONADOTRPHON 5500-I.U., 0000 I.U., 5000-I.U.
GONADOTRAFON LH 125-I.U., 250-I.U. 1000-I.U.
HCG 5000 I.U., 10,000 I.U. AMPULE
PREGNYL 1500 I.U., 5000 I.U. AMPULE



Reported Characteristics
Active Life: 8-12 hours
Drug Class: Synthetic estrogen/gonadotropin stimulator/anti-estrogen (Oral)
Average Reported Dosage: Men-400-600mg daily
Acne: Rare/light
Water Retention: Rare
High Blood Pressure: Rare
Strong gonadotropin stimulator/anti-estrogen
Aromatization: None
Liver Toxic: None noted


Cyclofenil administration is quite similar to using HCG and CLOMID together. It is an anti-estrogen and a gonadotropin/testosterone stimulator. It does so by occupying estrogen receptors (antagonist) with a much weaker estrogenic compound and by shutting down the negative feed-back within the hypothalamus-pituitary-testes-axis (HPTA). The male body actually can produce much more testosterone than it does. Simply said, it sees no reason to do so. When the hypothalamus senses adequate testosterone/estrogen levels, either naturally (endogenous) or unnaturally (exogenous) provided, it shuts off gonadotropin release from the pituitary/hypothalamus. Thus, leydig cells in the testes do not receive the signal to produce more testosterone.


Cyclofenil interferes with this negative feed-back/shut down production signal. This means that the hypothalamus – pituitary-testes-axis runs wide open to some extent, and more testosterone is produced. After about 5-6 weeks the hypothalamus figures this out and really shuts down the goodies production. Though some strength and mass resulted with Cyclofenil use, it was not noted as a great growth drug. Some “natural” (okay) bodybuilders have used it with some results as have older individuals. However, the chemically assisted lads have used it to kick start natural testosterone production and even as a “during cycle anti-estrogen” very successfully. Normally 200 mg was taken 2-3 times daily for 4-6 weeks, either starting the last 3 weeks prior to the end of an AAS cycle or directly following a cycle. The prior method being reported as more effective since about a week is necessary for Cyclofenil to become effective and provide results. Side effects commonly reported have been; light acne, elevated sex-drive (that is a side effect?), and hot flashes.


A note of interest; I have known several so-called natural bodybuilders who have utilized cyclofenil with a prohormone protocols in the United States with surprising results. This was effective for many reasons. Lower estrogen activity means a harder appearance. Testosterone both endogenous and exogenous combine resulted in significant total testosterone levels as well as a higher free testosterone level due to prohormones ability to uncouple testosterone from SHGB to some extent. The usual cycle consisted of 6 weeks of prohormones with 4 weeks of cyclofenil beginning week #5 of the prohormone cycle. Doing so extended the cycle 2 weeks and assured no post-cycle HPTA suppression to deal with for most athletes. Some athletes reported week #6-8 produced good secondary growth. This may have been due to an up- regulation of LH, FSH, and the conversion enzymes responsible for prohormones becoming testosterone in blood as well as decreased activity of estrogen. My concern would be the rush of estrogen post-cycle since cyclofenil only blocks estrogen receptors instead of suppressing estrogen production. In that example, for me personally, Arimidex would have been a wiser choice beginning week #5 also.

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Reported Characteristics
Active Life: about 8-12 hours
Drug Class: Synthetic estrogen/HPTA stimulator (Oral)
Average Reported Dosage: Men 50-100 mg daily
Water Retention: No
High Blood Pressure: Rare
Aromatization: None
Liver Toxic: Low, seldom reported.
Strong gonadotropin stimulator/mild anti-estrogen


Clomiphene is a synthetic estrogen clinically administered to help women ovulate. Bodybuilders, (male) following AAS cycles, seeking to jump start natural testosterone production (or those that were simply seeking a natural testosterone spike) have used this drug with great success. Clomiphene increases activity in the hypothalamus-pituitary-gonadol axis by stimulating the release of more gondotropin from the pituitary gland. Therefore, a higher/faster level of LH (luteinizing hormone) and FSH (follicle stimulating hormone) results. This creates a signal to the leydig cells in the testes which in turn produce more testosterone and sperm. Normally with Clomiphene this took 5-15 days. Most started with 50-mg twice daily for about 5 days, then reduced intake to 50 mg once a day for 5-10 more days. Due to Clomiphene providing a fast response time, I felt it was often beneficial to use a dosage of 100 mg total daily for 5 days, mid-cycle during longer AAS protocols. This drug was seldom utilized for longer than 15 days continuously simply due to it being unnecessary. The goal was to normalize testosterone production post AAS cycle as quickly as possible so as to minimize post-cycle strength and mass loss. Not create dependency. HCG was combined with Clomiphene (Clomid) sometimes, or Clomiphene was used after HCG administration. This is because Clomiphene acts by affecting the hypothalamus and pituitary (hypophysis) and regenerating the whole regulating system, while HCG only “imitates” LH, thus stimulating the leydig cells in the testes to produce natural testosterone. (*Also see HCG)

Some may wonder about Clomiphene being a synthetic estrogen. Yes, it is, but it works as an anti-estrogen. This is due to the fact that Clomiphene has a very low estrogenic effect. This means stronger and more active estrogen, such as those formed during the aromatization of many androgenic steroids, are blocked out of the receptor-site and less estrogenic activity results; less gyno, less water retention. Clomiphene was by no means as effective as Novladex or Proviron for estrogen suppression, but post-cycle it helped greatly.



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