Reported Characteristics
Active-Life: 10-12 hours
Drug Class: Diuretic (Oral)
Average Reported Dosage: 50-200mg (total) daily

Spironothiazid is a diureteic and an ALDOSTERONE antagonist. This means that it suppresses the water retaining actions of the hormone ALDOSTERONE while lowering water retention by lowering electrolytes (potassium, sodium, and calcium). The advantage of the combination is that the potassium absorption effect by the spironolactone can be mediated by the hydrochlorthiazide. Because of this, some of the potassium loss side effects can be avoided. This is probably a safer choice than LASIX though any diuretic use should be medically monitored. Thiazides also lead to a lower loss of calcium. Side effects are commonly due to imbalances in electrolytes and fluids. They can include irregular pulse rate, cramps, and light headiness. Since the drug has ANTIANDROGENIC quality (spironolactone), men can experience possible gyno, and impotence due to higher dosage and/or prolonged use. These were noted to not occur due to the brief administration period normally employed. According to available literature, it is best to avoid higher potassium intake during use. Dosages were usually 50-150mg a day for no more than 3 days. Dosages were divided into 2-3 daily dosages.

Aldactazide 25/15-MG TABS
Aldactazide 25/50-MG TABS
Spironothiazid 25/25-MG TABS



*The most important water store is in blood and the vascular system. Without adequate water in the vascular system blood volume is compromised, and if severe enough, the result is death. So this rates a big number one in the water store hierarchy. *The second on the big three list is muscle tissue. Water is required within all muscle tissues, both smooth and fibrous, to support life sustaining metabolic processes. *The last area of importance for water storage is subcutaneous (under the skin) areas. For those who have not been paying attention, this is the area that a bodybuilder wants to eliminate as much water from as possible the day of a show. The results are a “make-it or break-it” issue. So how do we do it? Piece of cake! (But not during contest dieting!) The key to subcutaneous water control depends upon control of the hormone Aldosterone. Obviously estrogen control is part of this hormone cascade action/reaction factor.

But, our main focus is salt and water control, so Aldosterone is the key. Beginning 15 days out from a show, an athlete should increase salt intake 20- 30%. This of course means salt intake was never reduced to begin with. The amount must remain reasonably high and steady each day. This creates an environment in which the body does not have to release Aldosterone. This causes salt to stay in muscle tissue and the subsequent attraction of water stores there. Also, the all important maintaining of the sodium-potassium pump is accommodated as well. (During diet phases, this also reduces catabolism.) During the 15 day period, water intake must absolutely remain high. 1.5-2.0 gallons daily is a base line in fact. This helps your body excrete any extra sodium, which of course it will, because Aldosterone secretion in the body has been controlled by elevated salt intake/water intake. The body will continue to dump all excess water and sodium as long as this is followed. On the Friday night before a Sunday show, the athlete stops water intake. The body thinks it will still get the 1.5-2.0 gallons of water daily and continues to excrete water at its normal rate. This causes a decrease in blood volume and of course muscle water volume. Remember the body’s water hierarchy? Well, as a survival response or reaction, the body gives up water from the area of least importance as a means of compensation. Yup, you got it. Subcutaneous water is pumped into blood and muscles. The result is vascularity, full muscle bellies, and paper thin skin. *It’s always a matter of working with, not against the body’s action/reaction factors to accomplish the greatest progress and /or results. *This works well with Creatine/Dextrose carb-loading also.

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Reported Characteristics
Active-Life: 8-12 hours
Drug Class: Steroid Biosynthesis Inhibitor (oral)
Average Reported Dosage: 500mg-2000mg Daily
Acne: None
Water Retention: None
High Blood Pressure: Unknown
Noted Comments: Powerful endogenous hormone inhibitor
Aromatization: None
Liver Toxic: High potential


Aminoglutethimide is normally prescribed for patients with breast cancer or Cushings Syndrome. In both cases patients suffer from catastrophic physiological decline. To a profound degree this is notably due to severe over production of endogenous glucocorticoids. The one glucocorticoid most readers would be familiar with is cortisol. Bodybuilders and strength athletes have often utilized this drug as a means of inhibiting the P-450 and aromatase enzymes. This obviously refers to inhibition of the two potential pathways in which susceptible AAS are converted into estrogens. The result of administration of aminoglutethimide is partial or complete inhibition of endogenous hormone biosynthesis. When this drug is introduced into the body it blocks the conversion of cholesterol into pregnenolone. Since this is the first step in all hormone biosynthesis: By blocking the conversion of C-19 androgens into C-15 estrogens it functions as an anti-estrogen. This is one of the obvious intents reported users had in mind. But by inhibiting the conversion of cholesterol into pregnenolone it also proportionately inhibits the endogenous biosynthesis of all hormones including androgens, estrogens, aldosterone, and cortisol. This brings the discussion to the second reason this drug use was commonly reported. Cortisol is a catabolic hormone that just loves to eat muscle tissue.


In fact it is a significant part of the equation that induces genetic limitations to muscular augmentation. Many authorities and athletes alike have realized on a few facts: (1) Less cortisol = less muscle catabolism (destruction) (2) Less estrogen = less fat accumulation and less gyno resulting in a leaner physique (3) Less aldosterone = less water retention and a harder appearance. But less conversion of cholesterol into pregnenolone = less endogenous testosterone. Most noted these effects were highly desirable since the absence of natural testosterone production was abundantly replace through AAS use. And it was generally acknowledged that the absence of competing hormones had a profound synergistic effect upon those the selfadministered. According to the available returns the average reported dosages were 500-750mg daily to inhibit excessive estrogen formation from AAS aromatization and 1000-2000mg daily for the purpose of cortisol inhibition. Most reported a scaled weekly dosage increase was most effective: Week #1 250mg 2xd, week #2 250mg 3xd, week #3 & 4 500mg 2xd. *My personal experience with this drug was that a 2 day on – 2 day off protocol worked best with fewer negative side effects. Of the many side effects is that the drug inhibits the body’s ability to react to inflammatory responses. This means it can prevent the body from inhibiting hemorrhaging (I do hope that no one cut themselves shaving!) and fight disease among other things. It can also make a bodybuilder a victim of CUSSING SYNDROME. Other side effects include: Sore joints from a reduction in glucocorticoids, reduced white/red blood cell counts, reduced platelet counts, and liver disease. The package insert states 2-7 250-mg tabs daily for treatment of CUSHINGS SYNDROME. CUSHINGS victims have a “much” higher cortisol/cortisone production than even the most over trained athlete. For this reason 500 mg-1000 mg daily total was considered more than enough for a chemically enhanced bodybuilder for cortisol/cortisone suppression, and 250 mg -500 mg sufficient for use as an anti-estrogen and aldosterone control drug during cycles. It was also noted that Aminoglutethmide was not be utilized for more than 4-6 weeks. At that point, the body responded by increasing production of ACTH and a whole new series of catabolic effects resulted.







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Reported Characteristics
Active-Life: 12-24 hours oral
Drug Class: Anti-estrogen/Aromatase enzyme inhibitor
Average Reported Dosage: Oral: 200-300mg daily; Injectable 100mg/1ml daily
Acne: Rare
Water Retention: None
High Blood Pressure: Rare
Liver Toxic: Not listed but unlikely
DHT Conversion: None
Decreases HPTA Function: No. (Actually increased HPTA activity)
Aromatization: None

Teslac is used as an anti-estrogen. Instead of acting as an estrogen antagonist (competing for estrogen receptor-sites), Teslac prevents the aromatization of androgens into estrogens by inhibiting the aromatase enzyme. Obviously this prevents or minimizes the negative effects of estrogen such as gyno, water retention, and female pattern fat deposits. Teslac is unique in that it is reputed to cause permanent irreversible suppression of estrogen production in males. Technically speaking, Teslac is an oral androgenic steroid related to testosterone. However, it has only very low androgenic and no anabolic effect. Medical use of Teslac is for treatment of breast carcinoma in women. For male athletes, it is probably the best anti-estrogen available, and it causes an increase in natural (endogenous) testosterone production. Teslac elevates natural testosterone production by influencing the HYPOTHALAMUS to stimulate the PITUITARY to release more gonadotropin. This in turn simulates the testes Leydig cells to produce more testosterone at a significantly increased level (Comparable to HCG). Unlike HCG, Teslac takes several days to cause this effect.

So administration over a prolonged period (several days) clinically considered necessary (under a doctors care). For this reason Teslac does “create” an improved anabolic/androgenic environment. Even so-called natural (like lifting tons of weight and drinking protein powders is natural) bodybuilders have employed benefits form this drug. *Plasma calcium elevation has been during administration of Teslac. Manufacturer inserts suggest a dosage of 250-1000mg daily. However, this is not reported as necessary for estrogen suppression. Normally 200-300mg daily divided into 2-3 doses was reported as quite effective. Some reported 100-mg of the injection form version was even more effective. Due to the high price of Teslac, many have stacked it with PROVIRON: Teslac-100-mg daily and Proviron 50-mg daily. Women users reported excellent hardening effects with few, if any, side effects such as virilization.

Side effects are very rare but could include nausea, vomiting, tongue infection, swelling and pain in arms and legs, prickling sensation and high blood pressure, as well as loss of appetite. As mentioned earlier, side effects were rare but women often skipped menstruation during use. This is probably rated as the most effective overall anti-estrogen available whether during or post cycle as well as for pre-contest overall hardening effects.


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Reported Characteristics
Active-Life: 64 hours
Drug Class: leutenizing Hormone (LH) – Gonadotropin
Average Reported Dosage: Men 2000-5000 i.u. injected every 5th day
Acne: Yes
Water Retention: Yes, HCG is a female hormone.
Liver Toxic: None
Aromatization: None, however due to HCG use testosterone levels increased and
aromatize was a potential side effect

HCG is used medically to positively influence ovulation in women, and to help produce estrogen. It is also utilized in fertility medicine to aid in ovulation. Male bodybuilders used HCG for another important reason. HCG is almost exactly the same amino acid sequence as Luteinizing Hormone (LH). LH is normally produced in the pituitary gland which is then circulated to the testes where it contacts the Leydig cells. The Leydig cells then produce androgens such as testosterone. Obviously this means so-called natural bodybuilders reported use of HCG to increase endogenous (natural) testosterone levels. According to some clinical studies this works so well that an injection of 1500-2000 i.u. of HCG has increased plasma testosterone levels 200-300% over normal levels. For those males who utilized high aromatizing AAS, HCG was a “partial” cure for restarting natural testosterone production either mid or post-cycles. When administering exogenous (occurring outside the body) androgens, such as AAS, the body’s endocrine system shuts down (partially of completely) natural androgen production in an attempt to maintain homeostasis.

This is simply because the hypothalamus senses the excess estrogens from AAS aromatization (and to a lesser degree elevated androgen levels) and signals the pituitary (hypophysis) to partially or completely stop producing LH and FSH (follicle stimulating hormone). Since the Leydig cells in the testes do not receive the signal from LH, they partially or completely shut down testosterone production. Sperm production is also reduced as a result of FSH downregulation. Because the testes are not producing androgens and/or sperm at their normal level, the testes shrink. This often causes a drop in libido too. Many heavy AAS users reported sexual dysfunction for a brief (Brief?) period post-cycle. So what did the men do about “the boy’s”? HCG injections act as a replacement for the LH normally produced by the pituitary gland which normally stimulates androgen and sperm production by the testes. When 2000-5000 i.u. of HCG was injected every 5th day for 2-3 weeks, mid or post-cycle, the testes began to function again.

Also an increase in total testosterone was realized and athletes often made some of their best gains at this point. It also helped to keep the “significant other” significantly happy! When HCG was administered beginning the last week of an AAS cycle and for an additional 1-2 weeks post-cycle, testes function normalized again and much of the common post- AAS cycle muscle mass and strength loss was avoided. However, our athletes were not out of the woods (with acceptable wood) quite yet. Earlier, I mentioned that HCG has been a utilized as a “partial cure” for the shut-down of the hypothalamus-pituitary-testes-axis (HPTA). HCG only “replaces” natural LH. The pituitary and hypothalamus part of the HPTA still sense no reason to produce gonadotropins and restore normal LH/FSH production. So ending HCG administration sometimes only brought on another crash. But staying on HCG for more than 3 weeks without at least a month off between HCG cycles could cause permanent gonadal dysfunction and/or a desensitizing of Leydig Cells. Male bodybuilders commonly used Clomid or Cyclofenil with HCG (*See Clomid for more info).

Available literature shows that Clomid stimulates the pituitary to release more gonadotropin so a quicker and elevated level of LH and FSH are realized. By following an AAS cycle with 2000-5000 i.u. HCG every 5th day for 2-3 weeks and ingesting Clomid for the last 10-15 days of HCG administration many athletes noted that muscle mass and strength losses post AAS cycle were significantly avoided. Many athletes also used Clenbuterol at this point. It should be noted that administration of HCG will increase plasma testosterone levels 200-300% or more. Therefore all of the negative side effects of testosterone injections can apply to a lesser degree.

A.P.I. 5000I,U., 10,000 I,U, 20,000 I,U, AMPULE
CHORAGON 1500 I.U., 5000 I.U. AMPULE
CHOREX 5000-I.U., 10,000 I.U. AMPULE
CHORON 10 1000 I.U., 10,000 I.U. AMPULE
GONADOTRPHON 5500-I.U., 0000 I.U., 5000-I.U.
GONADOTRAFON LH 125-I.U., 250-I.U. 1000-I.U.
HCG 5000 I.U., 10,000 I.U. AMPULE
PREGNYL 1500 I.U., 5000 I.U. AMPULE



Reported Characteristics
Active Life: 8-12 hours
Drug Class: Synthetic estrogen/gonadotropin stimulator/anti-estrogen (Oral)
Average Reported Dosage: Men-400-600mg daily
Acne: Rare/light
Water Retention: Rare
High Blood Pressure: Rare
Strong gonadotropin stimulator/anti-estrogen
Aromatization: None
Liver Toxic: None noted


Cyclofenil administration is quite similar to using HCG and CLOMID together. It is an anti-estrogen and a gonadotropin/testosterone stimulator. It does so by occupying estrogen receptors (antagonist) with a much weaker estrogenic compound and by shutting down the negative feed-back within the hypothalamus-pituitary-testes-axis (HPTA). The male body actually can produce much more testosterone than it does. Simply said, it sees no reason to do so. When the hypothalamus senses adequate testosterone/estrogen levels, either naturally (endogenous) or unnaturally (exogenous) provided, it shuts off gonadotropin release from the pituitary/hypothalamus. Thus, leydig cells in the testes do not receive the signal to produce more testosterone.


Cyclofenil interferes with this negative feed-back/shut down production signal. This means that the hypothalamus – pituitary-testes-axis runs wide open to some extent, and more testosterone is produced. After about 5-6 weeks the hypothalamus figures this out and really shuts down the goodies production. Though some strength and mass resulted with Cyclofenil use, it was not noted as a great growth drug. Some “natural” (okay) bodybuilders have used it with some results as have older individuals. However, the chemically assisted lads have used it to kick start natural testosterone production and even as a “during cycle anti-estrogen” very successfully. Normally 200 mg was taken 2-3 times daily for 4-6 weeks, either starting the last 3 weeks prior to the end of an AAS cycle or directly following a cycle. The prior method being reported as more effective since about a week is necessary for Cyclofenil to become effective and provide results. Side effects commonly reported have been; light acne, elevated sex-drive (that is a side effect?), and hot flashes.


A note of interest; I have known several so-called natural bodybuilders who have utilized cyclofenil with a prohormone protocols in the United States with surprising results. This was effective for many reasons. Lower estrogen activity means a harder appearance. Testosterone both endogenous and exogenous combine resulted in significant total testosterone levels as well as a higher free testosterone level due to prohormones ability to uncouple testosterone from SHGB to some extent. The usual cycle consisted of 6 weeks of prohormones with 4 weeks of cyclofenil beginning week #5 of the prohormone cycle. Doing so extended the cycle 2 weeks and assured no post-cycle HPTA suppression to deal with for most athletes. Some athletes reported week #6-8 produced good secondary growth. This may have been due to an up- regulation of LH, FSH, and the conversion enzymes responsible for prohormones becoming testosterone in blood as well as decreased activity of estrogen. My concern would be the rush of estrogen post-cycle since cyclofenil only blocks estrogen receptors instead of suppressing estrogen production. In that example, for me personally, Arimidex would have been a wiser choice beginning week #5 also.

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Reported Characteristics
Active Life: about 8-12 hours
Drug Class: Synthetic estrogen/HPTA stimulator (Oral)
Average Reported Dosage: Men 50-100 mg daily
Water Retention: No
High Blood Pressure: Rare
Aromatization: None
Liver Toxic: Low, seldom reported.
Strong gonadotropin stimulator/mild anti-estrogen


Clomiphene is a synthetic estrogen clinically administered to help women ovulate. Bodybuilders, (male) following AAS cycles, seeking to jump start natural testosterone production (or those that were simply seeking a natural testosterone spike) have used this drug with great success. Clomiphene increases activity in the hypothalamus-pituitary-gonadol axis by stimulating the release of more gondotropin from the pituitary gland. Therefore, a higher/faster level of LH (luteinizing hormone) and FSH (follicle stimulating hormone) results. This creates a signal to the leydig cells in the testes which in turn produce more testosterone and sperm. Normally with Clomiphene this took 5-15 days. Most started with 50-mg twice daily for about 5 days, then reduced intake to 50 mg once a day for 5-10 more days. Due to Clomiphene providing a fast response time, I felt it was often beneficial to use a dosage of 100 mg total daily for 5 days, mid-cycle during longer AAS protocols. This drug was seldom utilized for longer than 15 days continuously simply due to it being unnecessary. The goal was to normalize testosterone production post AAS cycle as quickly as possible so as to minimize post-cycle strength and mass loss. Not create dependency. HCG was combined with Clomiphene (Clomid) sometimes, or Clomiphene was used after HCG administration. This is because Clomiphene acts by affecting the hypothalamus and pituitary (hypophysis) and regenerating the whole regulating system, while HCG only “imitates” LH, thus stimulating the leydig cells in the testes to produce natural testosterone. (*Also see HCG)

Some may wonder about Clomiphene being a synthetic estrogen. Yes, it is, but it works as an anti-estrogen. This is due to the fact that Clomiphene has a very low estrogenic effect. This means stronger and more active estrogen, such as those formed during the aromatization of many androgenic steroids, are blocked out of the receptor-site and less estrogenic activity results; less gyno, less water retention. Clomiphene was by no means as effective as Novladex or Proviron for estrogen suppression, but post-cycle it helped greatly.



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Reported Characteristics
Active-Life: 4-6 hours
Drug Class: Anti-estrogen /competitive inhibitor (Oral)
Average Reported Dosage: 0.5-3.0 mg daily
Acne: No
Water Retention: None
Liver Toxic: Yes dosage dependent
Decreases HPTA function: No, the drug has been accredited with HPTA up-regulating

Arimidex is an anti-estrogen type drug. It is usually provided in 0.5 MG tabs. The drug works in a non-steroid form by inhibiting the aromatase enzyme which converts testosterone and other androgens into estrogen. This means that there is less estrogen to cause female pattern fat deposits, gyno, and water retention. In medicine, Arimidex is utilized to treat prostate cancer. In sports chemistry, the drug has been employed as a means of preventing excessive estrogenic side effects during AAS use and to aid in creating a harder appearing musculature for competitive bodybuilders. Unlike Nolvadex, which simply block estrogen receptor-sites, this drug prevents or reduces estrogen production. Though some estrogen presence is noted as necessary for AAS to reach full effectiveness, too much can cause a layer of fat, water retention, and breast tissue growth potentially with tumors called gynecomastia or bitch tits.

Arimidex has a 75-85% aromatization inhibition rate. Males who experienced excessive aromatization of AAS or who were extremely estrogen sensitive usually utilized a dosage of 0.5-3.0 mg daily. In fact, most realized excellent estrogen control with only 0.5mg/d (mg daily). Women usually showed excellent lean appearances (even in their legs) with 0.5-1.0 mg daily. Arimidex has a very short active-life so 0.5 mg dosages were often taken 2-6 times daily at equal intervals. Stacking 10-30 mg of Nolvadex with 1.0 mg of Arimidex has resulted in a near “0” estrogen activity situation regardless of the AAS protocol utilized. Directly following an AAS cycle, estrogen control has also become a problem (during periods intended for reestablishing HPTA function). In this case, the dosage was reduced from a higher starting dosage to a low dosage that was continued for 7-14 days after AAS discontinuance. This protocol was considered necessary to assure clearing of AAS induced estrogen build-up.

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Reported Characteristics
Active-Life: About 5-7 days
Drug Class: Androgenic/Anabolic Steroid (For injection)
Average Dosage: Men: 76-228-mg weekly; Women: 76-mg weekly (but should not use)
Acne: Rare
Water Retention: Rare, usually none
High Blood Pressure: Rare
Liver Toxic: Yes, highly toxic to kidneys also
Aromatization: None
DHT Conversion: None
Decreases HPTA function: Moderately


Parabolan was reported to be the most effective injectable anabolic/androgenic steroid available today. Real Parabolan provided users with a rapid build-up in strength and high quality muscle mass, while increasing fat burning (even when not dieting). The drug does not aromatize to estrogen or reduce to DHT. For this reason there was no noted water retention or feminizing effects. Parabolan sped up the metabolism as well. Due to the high androgenic quality, a hard, ripped, and vascular look with a dramatic increase in quality muscle mass resulted for all that reported use. This profile is what made Parabolan appear to be another all-purpose steroid. So what’s the problem? It was rare to find real Parabolan. Though the real product has a distinct odor, at least 95% seen was bogus. The real product is quite toxic to kidneys, so users mostly kept usage to 4-8 weeks and drank at least an additional gallon of water daily. Due to the high degree of potential toxicity dark urine and even blood in urine can result from high, prolonged use (as can prostate enlargement due to the drug’s structural similarity to DHT).


Parabolan was commonly stacked with growth hormone, thyroid, and/or Clenbuterol during diet periods for the synergistic effects. A simple stack of 20-30-mg Oxandrolone daily and 152-228-mg Parabolan weekly consistently brought excellent high quality strength and muscle mass gains while decreasing body fat. Women should not use Parabolan due to a very high virilizing effect. But many competitive female athletes still did. Some stacked 76-mg of Parabolan every 5-8 days with 80-120 mcg of Clenbuterol and either 15-20-mg of Winstrol tabs or 10-15-mg of Oxandrolone tabs daily. I know of one female who is suffering from most of the possible side effects: missed periods, hair loss on the scalp, serious sex-drive (you should see her on an exercise bike) and clitoral hypertrophy. Facial hair growth can also result also.

Males usually obtained excellent results with 76-mg every 2-3 days.(Though some doubled this amount unnecessarily)




Reported Characteristics
Active-Life: About 24 hours
Drug Class: Androgenic/Anabolic Steroid (For injection)
Acne: Yes
Liver Toxic: Very
Water Retention: Low-None
High Blood Pressure: Rarely
Aromatization: None

Metribolone is the trade name for the drug Methyltrienolone, which is the most potent AAS produced. It is actually an orally active form of Trenbolone chemically altered into a 17-alkylated compound. Obviously, this means serious liver toxicity. In fact, even at microgram dosages, (1milligram = 1,000 micrograms) it is 15-20 times more toxic than Anadrol-50. That is toxic! Though orally active this AAS is provided in vials and meant for injection use only. But in truth, Metribolone is about 40-50 times more androgenic than Methyltestosterone, so a little goes a long way. The drug is fairly resistant to DHT conversion but does bind easily to scalp and prostate androgen receptor sites. In clinical research, Metribolone is used to determine receptor-site affinity / displacement. Let me explain that. Metribolone is a very powerful androgen receptor-site stimulator and antagonist. I doubt there is any AAS more powerful. Since it binds so strongly to the receptor-site, researchers use the drug to see if other drugs can dislocate it, or for comparison. Not even Deca can kick it out of receptor-sites! Metribolone is highly resistant to binding proteins such as SHBG. This means it remains highly active in the blood. If you recall, about 97-99% of our testosterone is in a “bound” state and only the remaining 1-3% is active or free. Only free or active androgens can fit into receptor-sites and trigger the anabolic mechanism.

Some AAS are more resistant to these binding proteins than others. And Metribolone is the most resistant of all. Like I said, a little goes a long way. As example consider this for a moment. If all other factors of potency were equal, 1mg of unbound/free Metribolone would have the same activity as 97-100mg of testosterone due to the effects of binding proteins. *Metribolone is like “Super Parabolan”. The few individuals I know who have tried it reported the stuff was nothing short of amazing. This is an injectable form of course. Since Metribolone has a brief active-life, daily injections were reported as necessary. With mega-dosage use liver damage is not just a high concern, it is a fact. Since this drug is like “Super Parabolan”, all negative side effects, results, and uses of the two drugs are interchangeable for the most part, with the obvious exception of reported dosages. I have seen only a few vials of the SP Labs product available on the black market as of yet. However, there is an injectable around that claims to be made by Denkall. It is an underground lab product actually and the mcg/ml is suspect as is sterile factors. Personally, I did/would not use the drug. It just amazes me that it exists.